GRP-093 Implementing and Improving Medicines Reconciliation on Admission at North Bristol NHS Trust (NBT): Abstract GRP-093 Table 1

Improving the accessibility of trust guidelines for junior doctors at North Bristol NHS Trust

Medicine is becoming increasingly protocol driven. This provides a standardised format for doctors to deliver best practice, especially in the acute setting. 40 junior doctors were asked to locate three Trust guidelines: venous thromboembolism (VTE) prophylaxis; antibiotic prescribing; and management of upper gastrointestinal bleed (UGIB). For each doctor the time taken and number of mouse clic...

Improving medicines reconciliation rates at Ashford and St. Peter's Hospitals NHS Foundation Trust

Medicines reconciliation is integral to patient safety, symptom control and reducing patient anxiety. During a 3-month period on the respiratory ward at St. Peter's Hospital, 54% of drug charts were not reconciled with pre-admission medicines at the point of discharge for admissions up to 17 days. Only 18% were reconciled within 24 hours of admission. 50% of drug charts were missing 0-2 pre-adm...

Improving the quality of discharge summaries for elective surgical procedures at North Bristol NHS Trust

Following elective surgical procedures, hospital discharge summaries are essential in the handover to primary care. The information provided varies between institutions and is highly user-dependent. Various interventions have focused on improving information transfer to patients and primary care physicians including the development of electronic templates, electronic transmission to primary car...

Grp 78 , Grp 94 , and Grp 170 interact with 1 - antitrypsin mutants that are retained in the endoplasmic reticulum

Schmidt, Bela Z., and David H. Perlmutter. Grp78, Grp94, and Grp170 interact with 1-antitrypsin mutants that are retained in the endoplasmic reticulum. Am J Physiol Gastrointest Liver Physiol 289: G444–G445, 2005. First published April 25, 2005; doi:10.1152/ajpgi.00237.2004.—In 1-antitrypsin ( 1-AT) deficiency, a mutant form of 1-AT polymerizes in the endoplasmic reticulum (ER) of liver cells r...

Ecological Archives E 092 - 093 - A 1